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In addition, SIRT1 deacetylates FOXOs and stimulates FOXO-dependent antioxidant [such as catalase (CAT), manganese superoxide dismutase (MnSOD) and thioredoxin] expression to eliminate ROS in endothelial cells, and prevent endothelial dysfunction [78-80]

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Furthermore, translational challenges such as tissue targeting (e.g., bloodbrain barrier penetration), metabolic stability (e.g., optimization of CoQ analogs), and the need for predictive biomarkers underscore the importance of continued mechanistic studies to refine therapeutic strategies exploiting these emerging ferroptosis defense nodes